Are You Guaranteeing That Your Children Will Get Cancer?

If you’re smoking Swisher Sweets, you are. It has nothing to do with tobacco, or nicotine. It also doesn’t matter whether you’re a Man or Woman. There’s a hidden man-made mutagen called DDT in those little cigars that is zapping either your eggs or sperm and causing permanent genetic damage that will be passed on to your kids, and then their kids, causing them a very high risk of getting cancer and developing obesity and diabetes no matter how much they try to live healthy lives. They will have started out damaged, and they will lead damaged lives. 

What – you think the government wouldn’t allow that to happen? Especially since Swisher Sweets are marketed to young men and women who haven’t even started a family yet? Especially since DDT is banned worldwide in any consumer product?

Wrong. The FDA officially doesn’t care. They don’t regulate pesticides in tobacco products. Not DDT, not anything else you see in the data below. They never have, and they don’t want to. They’re hell on wheels when it comes to mint and fruity flavors, but not a peep about genetically mutated children. And they know this is happening – for sure.

So check the data. See that DDT in the Swisher Sweets? That’s a huge concentration – 0.816 Mg/Kg – over 700 times what is found in any food or consumer product, and more than enough to attack and destroy and any baby unfortunate enough to be conceived and born to a Swisher Sweets smoking Mom or Dad. 

Want a little published scientific proof of what I’m saying? Here’s just one recent study. You can Google DDT and “genetic” or “fetal” and see dozens more published research studies. But really, how much evidence is enough to demonstrate that inhaling DDT isn’t a good idea?

Epigenetic Inheritance: Intergenerational Effects of Pesticides and Other Endocrine Disruptors on Cancer Development

“Studies in both animal models and human cohorts have linked exposure to pesticides and other environmental toxicants to defects in the parental germline.

For instance, in utero exposure to DDE, a DDT metabolite, impairs male fertility and causes epigenetic alterations in the male germ-line in rats. Other pesticides have similar effects: prenatal dioxin induces sperm epimutations and disease in subsequent generations.

Studies in human cohorts support the findings in animal models and report an association between pesticide exposure, including DDT, and sperm quality. The female germline and reproductive health have also been shown to be sensitive to the endocrine disrupting action of pesticides, with long-term adverse effects to offspring.

Accordingly, epidemiologic studies have linked parental occupation—and associated pesticide exposure—to cancer and other diseases in their children.

This environmentally induced disease risk has been shown to be transmitted to the offspring via epigenetic mechanisms through both the female and male germ-lines. Although most of the evidence for this mode of disease inheritance comes from maternal exposures in pregnancy, we and others have shown that paternal exposures in the pre-conception window are also important in determining disease outcomes in the offspring.

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