Recently on this blog I’ve posted what I believe is solid evidence that Coca Leaf tea can probably be used as-is, right out of the “Mate de Coca” bag from Peru or Bolivia, to successfully treat inflammatory obesity and many chronic conditions underlying congestive heart failure.
This would NOT be the Coca Leaf tea that has been ‘sanitized’ for export to the US – the “de-cocainized” stuff that the US government so graciously allows in. I’m talking of course about the pure, natural leaves of the Coca plant, produced, packaged and widely consumed in Peru and Bolivia. There is where I believe that a very simple but effective approach could be devised to begin validating Coca Leaf as a medicinal herb of the highest order.
When you think about it there is already remarkable evidence right in front of our eyes that Coca Leaf is probably a powerful, natural medicine for a range of health issues – it’s just one of those things hidden in plain sight.
Up to 500,000 travelers to the Andes in the past couple of years are estimated to have used Coca Leaf tea successfully for altitude sickness, and of course for countless generations the indigenous people of the mountains have seen this benefit too. (They have also, in the absence of western disease and culture, often lived long and productive lives.) There is solid evidence going back as far as western records go that the powers of Coca Leaf in effectively treating Altitude Sickness have been proven beyond dispute.
So, we know that the Divine plant is useful for altitude sickness. We also know that it’s effective because of its broad action on multiple body systems – heart, lungs, muscle efficiency, metabolism, brain function, and oxygen use. All of the same body systems, incidentally, involved in inflammatory conditions that lead to obesity and congestive heart failure.
But because so many people know from experience that Coca Leaf is so good at relieving altitude sickness- then it’s easy to miss the follow-up question – “what else could Coca Leaf tea be doing?”
So let’s zero back in on inflammation. It’s a very interesting topic in the medical literature because it is so often treated as the baseline, the underlying condition that is simply there. Chronic low-level inflammation is simply the way things are for lots of people. Doctors seem to start with inflammation as a given – they “give you something for it” and then go on and ‘treat’ the conditions that have arisen from the underlying inflammation, which so often remains despite “medications”.
A little while ago I posted an opinion on a forum elsewhere regarding chronic low-level inflammation, industrial foods and obesity and that post provoked a lot of “fat hate” comments like “Oh, a new excuse for being a fat pig”, and “the only thing inflamed is their greedy gut” and other such slobbering wit.
Maybe it’s difficult for people, even those affected, to visualize whole body inflammation. Most of us can picture an inflamed toe, or a sore throat, but what does who body inflammation look like?
Well, if it’s low-level it isn’t going to be dramatic – like an open sore. And if it’s whole body then nothing will be disproportionate – like a 3X normal toe.
The idea of chronic low-level whole body inflammation has always made sense to me because I link it to my own experiences with cuts and sores during my life. Depending on the cut or sore, and whether or not it leads to infection, there are different levels of the body’s own attack on the cut or sore. It always involves sore, pink flesh – responding to the injury with blood and body fluids that swell cells and flood the area with healing bio-chemicals. But – the clue to me is “Pink”. When the area around a sore or wound turns Pink then at that moment it is always in the first stages of inflammation.
So then I mentally translate that very early stage pinkness to a whole body situation and what I get is slightly-to-definitely swollen, slightly to definitely “Pink” people (whether their natural racial color is black, Brown or White). There’s just a swollen quality to them, and they move as if they are in pain.
I see a lot of those people as I move around my community and elsewhere. I see a lot of people with chronic low level whole body inflammation as a baseline in their lives, which then creates a short but deadly list of diseased conditions like diabetes and heart disease. And I see people who I sincerely believe could be helped by a few cups of tea a day from the Divine Coca plant.
So I have to ask – what if it were possible to conclusively prove that you could control chronic low-level whole body inflammation with either a few cups of Coca leaf tea every day, or with safe doses of a natural extract of Coca Leaf such as the 1890’s “Vin Mariani”? As you read the opening paragraph of the summary of the following research, ask yourself what if the conditions that the authors identify with chronic low-level inflammation could be controlled so easily and naturally? Who knows if Coca Leaf offers a cure for chronic low-level inflammation especially if the “cause” of the inflammation is a combination of environment, genetics, and behavior.
I’m sure that new fortunes will be made in Peru, Bolivia and other Andean countries by those who dare to reclaim their heritage and use the Coca Leaf for natural healing of inflammatory disease in clinics and spas throughout the Andes. Later I believe that this will happen in the Pacific Northwest and the Rocky Mountains, and in other areas of the world where we already know from the historical record that Coca grows well.
Chronic low-level inflammation isn’t a mysterious topic, and the fact that it underlies a lot of disease process, and might be treated or cured by Coca Leaf, makes it worth asking the question.
With the role of Coca Leaf chronic low level inflammation in mind it’s interesting to note the following excerpts of a research paper on Cytokine levels in different populations. As I understand it, Cytokines are a family of small proteins circulating in the blood and involved in cell signaling, some of which are used by the body to guide inflammatory processes to wounds, infections, and the like. They are reliable markers of low-level widespread inflammation, among other things, and this research indicates that there are lots of reasons for different people to suffer from the condition. Parenthetically I have to wonder what the results would have been if the following study had been able to include a sample of recent immigrants of Andean origin?
Plasma Cytokine Levels in a Population-Based Study: Relation to Age and Ethnicity
Raymond P. Stowe, M. Kristen Peek, Malcolm P. Cutchin, and James S. Goodwin
E-Pub Info: J Gerontol A Biol Sci Med Sci. 2010 Apr;65(4):429-33. doi: 10.1093/gerona/glp198. Epub 2009 Dec 16.
“Inflammation is believed to contribute to the onset of many age-related diseases as evidenced by a variety of medical studies linking proinflammatory cytokines to Alzheimer’s disease, Parkinson’s disease, amyotrophic lateral sclerosis, and multiple sclerosis. Much attention has been paid to interleukin (IL)-6 because of its association with cardiovascular disease, the leading cause of death. IL-6 is associated with a broad spectrum of age-related illnesses, chronic stress, and functional disability in older adults. IL-6 is also a strong inducer of C-reactive protein (CRP) by the liver, and both IL-6 and CRP are important in the development of cardiovascular disease. IL-6 and CRP also play a pathogenic role in a number of diseases associated with disability in older adults, such as arthritis, osteoporosis, and depression among others.”
“Studies of older humans have reported age-related increases in proinflammatory cytokines, but the switch from the inflammatory burst that resolves following an infection or injury to the chronic elevation encountered in many older adults is not well understood. Several investigations have indicated that there is an age-related increase in circulating IL-6, which has been called a “cytokine for gerontologists”. However, some studies have found no changes with age. Similarly, tumor necrosis factor (TNF)-α, a cytokine that is involved in septic shock, was reportedly increased in some studies but not others.”
“Anti-inflammatory mediators, such as IL-10 and interleukin-1 receptor antagonist (IL-1ra), may also be important in the aging process because they counteract proinflammatory cytokines. With regards to IL-10, few studies have measured circulating levels of this cytokine, but there have been reports that indicated no change occurs with aging. Reports have also shown an age-related increase in the IL-1ra. Altogether, the discrepancies regarding these cytokines mostly likely relate to variations in age and sample size.”
“Besides age, cytokine levels may also be influenced by ethnicity. Plasma levels of IL-8 and granulocyte colony-stimulating factor were elevated in African Americans compared with Caucasians, and TNF-α has been reported to be higher in nonobese Mexican Americans compared with non-Hispanic whites. Because there is little other information on circulating proinflammatory cytokine levels and ethnicity, our goal was to investigate plasma levels of circulating cytokines in relation to ethnicity as well as age in a large population-based study. We found age-related differences in proinflammatory cytokines as well as significant differences in circulating cytokine levels between Mexican Americans, non-Hispanic whites, and non-Hispanic blacks.”
“Because our study population was tri-ethnic in nature, we analyzed the data accordingly and found significant associations between cytokine levels and ethnicity. The highest levels of proinflammatory markers were found in either non-Hispanic whites or blacks. Interestingly, it has been proposed that the health status of Hispanics in the Southwestern United States is comparable more so with whites than with blacks despite the fact that socioeconomically Hispanics are more similar to blacks than the more advantaged whites; this has been aptly named the “Hispanic Paradox”. Our results support this concept, which demonstrates that, collectively, lower levels of inflammatory cytokines were found in Hispanics compared with whites or blacks. Notably, the lowest inflammatory levels were found in foreign-born Hispanics. We have previously proposed that protective measures (eg, acculturation) may in part underlie the differences between foreign-born and US-born Hispanics because we found that increasing years in the United States was associated with increasing IL-1ra levels among Hispanic women at 22–24 weeks of pregnancy. Further research is needed to determine the mechanisms underlying the differences in cytokine profiles between foreign-born and US-born Hispanics.”
“One potential explanation for the ethnic variations in cytokine levels is differences in cytokine gene polymorphisms. Allelic variations in the regulatory regions of inflammatory cytokine genes have been shown to affect the expression of some cytokines. Several studies have focused on IL-6 because of its biological importance and have demonstrated that the G/G IL-6 genotype, which results in high IL-6 production, is predominantly found in blacks. It has been hypothesized that the dissimilarities in cytokine gene polymorphisms may contribute to the differences in inflammatory responses and cancer incidence and mortality in blacks. Additionally, obesity was a significant determinant of CRP levels in non-Hispanic blacks, and BMI was significantly higher in non-Hispanic blacks than either non-Hispanic whites or Hispanics (data not shown).”
“In summary, our results confirm and extend other studies demonstrating age-related increases in circulating proinflammatory cytokines. In addition, we have shown ethnic differences in cytokine levels, and to our knowledge, this is the first study demonstrating ethnic differences in proinflammatory and anti-inflammatory cytokine profiles in large population-based study. Future studies are needed to determine the epigenetic link between inflammation and ethnicity.”
Editor’s Afterthought: When it’s so clear that chronic low-level inflammatory processes are at work underlying so much disease, is there any good reason that pure, natural Coca Leaf sourced directly from growers in Peru and Bolivia by legal means shouldn’t be tested for potential health benefits?
Even if Coca Leaf were simply helpful, without causing any harm, Coca Leaf could form the core of therapies at spas and clinics. And incidentally, these Spas and Clinics should be free to grow their own Coca Leaf or to contract with any indigenous person or group to grow their Coca Leaf for them.