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Thoughts On Coca, Cannabis, Opium & Tobacco – Gifts Of The Great Spirit

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Swisher Sweets Cause Irreversible Neurological Damage To Black & Brown Babies

This post is about a crime being committed right now, today and every day, against hundreds of thousands of Black and Brown living and unborn babies. Their lives are in grave danger from nerve gas and genetic toxins in those fruity little Sweets. Many babies and children have already been irreparably damaged. But those young Black and Brown lives don’t matter, not to Swisher Sweets. This is a WAY profitable product, made with the worst, most contaminated tobacco waste. They market directly to Black and Brown young people, especially girls and women, using every evil little “targeted marketing” trick in the tobacco industry’s book. The facts prove that they knowingly and without regard to human life produce an unreasonably dangerous, life-threatening product that has especially toxic impact on babies with Black or Brown genetics.

Pretty stark, I know, but check this table that shows you the concentration of DDT found in Swisher Sweets when I had them tested by a highly competent lab. There are many more neurotoxic fungicides and insecticides in good old fruity minty fun and funky Swishers, as you can see, but let’s just focus on the DDT.

That DDT concentration you can see in the table – 0.816 ppm – is 700+ times greater than the concentration of DDT in any 2019 food or other consumable product. That’s a HUGE problem for a fetus whose Mommy is inhaling that DDT 50-100 times a day or more. Many young Black and Latina women actually believe the street myth that smoking Swishers is safer than smoking cigarettes so they pass their entire pregnancy inhaling these fruity little sacks of nerve poison.

DDT has been banned for any use except extreme mosquito control for generations, and for plenty of good reasons. It’s still the best killer the chemical industry has ever come up with, bar none, and growers worldwide long to be able to use it again. But alas, they can’t. Unless they’re the tobacco industry, and then it’s OK. So – here it is, freshly applied to tobacco and being inhaled by 3 million+ young Swisher Sweets smokers, predominantly Black and Latinx, over half female.

How many Swisher smokers are pregnant today? We know that only 25% of them will stop smoking during pregnancy. There are communities full of damaged babies of Black & Brown smokers and nobody knows why so everybody just seems to accept it – that’s just the way it is if you smoke. Maybe you can see, after you’ve looked at this evidence, the real reason why so many babies of Black and Brown smokers are born damaged beyond belief. And why it doesn’t have to be that way.

The research evidence, quoted and linked in-depth below, is overwhelming that even the tiniest exposure to DDT at the right point in fetal development can be catastrophic. That’s true of other neurotoxins in Swishers, each with its own special kind of damage. After all, the whole purpose of these neurotoxins is to kill and destroy millions of tiny bugs in a field with just a few molecules of the neurochemical for each tiny life. These chemical companies are the direct descendants of Nazi nerve gas manufacturers and they have not changed their attitude toward “sub-humans” one little bit.

Babies in the womb are being drenched with these neurotoxins through their mother’s blood, across the placenta, straight to their emerging brain, glands and nervous system. Their effects at the molecular level are just being documented, but they are terrible and permanent.

The US and world governments have quite deliberately never tested for these neurochemicals although they spend hundreds of millions of taxpayers money trying to convince people that smoking is bad. Any high-paid government flunky care to really say why? They all know – Agriculture, Justice, the EPA, the FDA, the CDC. The industry has the system rigged at every level of government so it is exempt from regulation anywhere, anytime for this beyond-criminal behavior.

Aren’t these crimes against humanity? Against the unborn? Could they be genocide? Gassing babies ought to qualify, don’t you think maybe? See what the research says about gassing babies just below the data table showing you what they’re being gassed with.

If this table is too small check the one above – and I’ve also included full-size Swisher Sweet & Marlboro tables, without the little Sweetie, at the end of this post.

Current Opinion in Pediatrics

2008 Apr; 20(2):191-7.

doi: 10.1097/MOP.0b013e3282f60a7d.

https://pubmed.ncbi.nlm.nih.gov/18332717/

Pesticides and child neurodevelopment

Recent studies on in-utero exposure to the organochlorine pesticide dichlorodiphenyltrichloroethane and its breakdown product, dichlorodiphenyldichloroethene, indicate that exposure is associated with impaired child neurodevelopment.

Basic Clinical Pharmacological Toxicology

2008 Feb;102(2):228-36.

doi: 10.1111/j.1742-7843.2007.00171.x.

Pesticide toxicity and the developing brain

https://pubmed.ncbi.nlm.nih.gov/18226078/

We present the work of the CHAMACOS study, a longitudinal birth cohort study of Mexican-American children living in the Salinas Valley of California. In this study, we investigated the relationship of children’s neurodevelopment with maternal dichlorodiphenyltrichloroethane and dichlorodiphenyldichloroethylene serum levels, as well as prenatal and child organophosphate urinary metabolite levels. We observed a negative association of prenatal dichlorodiphenyltrichloroethane exposure and child mental development.

Life Sciences

2016 Jan 15;145:255-64.

doi: 10.1016/j.lfs.2015.11.006. Epub 2015 Nov 5.

Potential role of organochlorine pesticides in the pathogenesis of neurodevelopmental, neurodegenerative, and neurobehavioral disorders: A review

https://pubmed.ncbi.nlm.nih.gov/26549647/

The growing body of evidence has demonstrated that prenatal exposure to organochlorines (OCs) is associated with impairment of neuropsychological development. It has been suggested that maternal exposure to OCPs results in impaired motor and cognitive development in newborns and infants. Moreover, in utero exposure to these compounds contributes to the etiology of autism. 

Indian J Med Res. 2016 Jun; 143(6): 685–687.

doi: 10.4103/0971-5916.191922

Organochlorine pesticides exposure & preterm birth

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5094107/

This study confirms the association between maternal serum concentration of OCPs and preterm labour, and also reports an increased mRNA expression of inflammatory pathway genes such as TNF-α and COX-2, which have not been correlated with OCPs levels in previous studies concerning preterm birth aetiology. Even if COX-2 and TNF-α cannot be considered as direct biomarkers for pesticide exposure, since these are involved in all inflammatory processes that contribute to the onset of PTB, a possible gene-environment interaction may occur. The association between OCPs with mRNA expression of TNF-α gene should be further investigated.

Toxicology

2013 May 10;307:136-45.

doi: 10.1016/j.tox.2012.06.009. Epub 2012 Jun 21.

Toxic effects of pesticide mixtures at a molecular level: their relevance to human health

https://pubmed.ncbi.nlm.nih.gov/22728724/

The toxicological effects of low-dose pesticide mixtures on the human health are largely unknown, although there are growing concerns about their safety. The combined toxicological effects of two or more components of a pesticide mixture can take one of three forms: independent, dose addition or interaction. 

Not all mixtures of pesticides with similar chemical structures produce additive effects; thus, if they act on multiple sites their mixtures may produce different toxic effects. The additive approach also fails when evaluating mixtures that involve a secondary chemical that changes the toxicokinetics of the pesticide as a result of its increased activation or decreased detoxification, which is followed by an enhanced or reduced toxicity, respectively. 

This review addresses a number of toxicological interactions of pesticide mixtures at a molecular level. 

Examples of such interactions include: 

  • the postulated mechanisms for the potentiation of pyrethroid, carbaryl and triazine herbicides toxicity by organophosphates; 
  • how the toxicity of some organophosphates can be potentiated by other organophosphates or by previous exposure to organochlorines; 
  • the synergism between pyrethroid and carbamate compounds and 
  • the antagonism between triazine herbicides and prochloraz. 

Particular interactions are also addressed, such as:

  • pesticides acting as endocrine disruptors, 
  • the cumulative toxicity of organophosphates and organochlorines resulting in estrogenic effects, and 
  • the promotion of organophosphate-induced delayed polyneuropathy.

Current Opinion jn Pediatrics

2008 Apr;20(2):184-90.

doi: 10.1097/MOP.0b013e3282f56165.

Prenatal tobacco smoke and postnatal secondhand smoke exposure and child neurodevelopment

https://pubmed.ncbi.nlm.nih.gov/18332716/

Prenatal tobacco and postnatal secondhand smoke exposure is consistently associated with problems in multiple domains of children’s neurodevelopment and behavior.

The literature on both prenatal and postnatal exposure is remarkably consistent in showing associations with increased rates of behavior problems, including irritability, oppositional defiant behavior, conduct disorders and attention deficit hyperactivity disorder.

Environ Health Perspect

2014 Oct;122(10):1103-9.

doi: 10.1289/ehp.1307044. Epub 2014 Jan 23.

Neurodevelopmental disorders and prenatal residential proximity to agricultural pesticides: the CHARGE study

https://pubmed.ncbi.nlm.nih.gov/24954055/

Conclusions: This study of ASD strengthens the evidence linking neurodevelopmental disorders with gestational pesticide exposures, particularly organophosphates, and provides novel results of ASD and DD associations with, respectively, pyrethroids and carbamates

Environ Health Perspectives

2017 May 25;125(5):057002.

doi: 10.1289/EHP504.

Prenatal Residential Proximity to Agricultural Pesticide Use and IQ in 7-Year-Old Children

https://pubmed.ncbi.nlm.nih.gov/28557711/

This study identifies potential relationships between maternal residential proximity to agricultural use of neurotoxic pesticides and poorer neurodevelopment in children.

Proc Natl Acad Sci U S A

2019 Sep 10;116(37):18347-18356.

doi: 10.1073/pnas.1903940116. Epub 2019 Aug 26.

Prenatal exposure to organophosphate pesticides and functional neuroimaging in adolescents living in proximity to pesticide application

https://pubmed.ncbi.nlm.nih.gov/31451641/

OP exposure was associated with altered brain activation during tasks of executive function.

Environ Health Perspect

2018 Apr 25;126(4):047012.

doi: 10.1289/EHP2580.

Prenatal Organophosphate Pesticide Exposure and Traits Related to Autism Spectrum Disorders in a Population Living in Proximity to Agriculture

https://pubmed.ncbi.nlm.nih.gov/29701446/

Conclusions: These findings contribute mixed evidence linking OP pesticide exposures with traits related to developmental disorders like ASD. Subtle pesticide-related effects on ASD-related traits among a population with ubiquitous exposure could result in a rise in cases of clinically diagnosed disorders like ASD

Environ Research

2016 Oct;150:128-137.

doi: 10.1016/j.envres.2016.05.048. Epub 2016 Jun 6.

Residential proximity to organophosphate and carbamate pesticide use during pregnancy, poverty during childhood, and cognitive functioning in 10-year-old children

https://pubmed.ncbi.nlm.nih.gov/27281690/

Conclusions: Residential proximity to OP and carbamate pesticide use during pregnancy and both household- and neighborhood-level poverty during childhood were independently associated with poorer cognitive functioning in children at 10 years of age.


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Epileptic Seizures, Autism, Dementia/Alzheimer’s, Cannabis … And Coca Leaf Tea?

First, A Short Summary:

  1. Researchers are “just discovering” that Cannabis can control Epileptic seizures (Most recent New England Journal of Medicine)
  2. Researchers have not yet looked at Cannabis for use in Dementia/Alzheimer’s, even though seizures are common and are a leading cause of death in Dementia/Alzheimer’s. (Update 6/19/17) – this has just changed – see this on a new German-Israeli study)
  3. Maybe that’s because while Congress has committed some $5.4 billion this fiscal year to cancer research, about $1.2 billion to heart disease and $3 billion to research on HIV/AIDS, research funding for Alzheimer’s is “only” $566 million. Clearly that’s just not enough for researchers (mostly Big Pharma employees) to look into Cannabis as a treatment.
  4. On a related note, California researchers have just shown that a sleeping sickness drug developed in 1916 can reverse Autism in children; unfortunately, the test group was only 10 kids, and 5 of them were given placebos, and the researchers had to go $500,000 into debt to run the study. Evidently kids with Autism aren’t a big enough deal for Congress. Thoughtful of those researchers to care enough to go ahead though.
  5. And to wrap all this up in a neat little package, I figure it would cost well under $100,000 to show that Coca Leaf can not only control Epileptic seizures (as already well-known and demonstrated in 1881), but probably also Dementia/Alzheimer’s seizures – not even a diagnosed disease in 1881.
  6. So, a drug from 1916 is now “discovered” to cure Autism, after decades of high-dollar research into “new” cures. And Cannabis is discovered to cure Epilepsy, after more decades of research into “new” cures. And an 1881 proven cure for both epilepsy and likely for Dementia/Alzheimers, among many other killer diseases, is illegal. Go figure.

The Full Story

For several years researchers have been zeroing in on Cannabis as a source for potent medicines in treating & preventing epileptic and other kinds of seizures. The latest findings, published May 25, 2017 in the New England Journal of Medicine, showed that @ 40% of those treated with a CBD-based medicine experienced dramatic improvement in seizure intensity and frequency.

So, let’s put this together with an interesting association between seizures and Dementia/Alzheimer’s. There is plenty of research on this association. Here’s just one example.

“Of the degenerative disorders, Alzheimer’s dementia and amyloid angiopathy are known major causes of seizures. Advanced Alzheimer’s disease has been identified as a risk factor for new-onset generalized tonic-clonic seizures in older adults. It is associated with a 10 percent prevalence of seizures, particularly late in the illness. An increased prevalence of seizures also has been documented with other types of dementia.

So I suppose that it would make sense to investigate whether Cannabis-derived medicines, or perhaps the right strain of Cannabis itself, could be useful in controlling or preventing seizures in Dementia/Alzheimer’s, especially in late-stages of the disease when seizures are a known killer.

I’m sure that researchers are already drafting multi-million dollar grants to study exactly that.

So far, so good. In spite of decades of “Killer Weed” propaganda it looks like scientific minds are finally rising above the lies and finding that, consistent with centuries of well-established knowledge, the natural medicine Cannabis can be helpful in dealing with killer seizures better and with less risk of harm than pharmaceuticals.

But wait! If centuries of medical knowledge regarding the efficacy and safety of Cannabis are now appearing as “new findings” in prestigious medical journals, why not take a look at centuries of medical knowledge regarding the safety and efficacy of Coca Leaf in the same area?

Hmmmm. Could it be that a cup or two of Coca Leaf tea a day might be helpful to people with Dementia/Alzheimer’s – at least in preventing seizures, if not in other ways too. Let’s see. Who would know?

Well, there is a little book entitled “Erythroxylon Coca”, written by By W.S. Searle, MD and published in New York in 1881. (Dr. Searles book is included in its entirety in my ebook “Coca Leaf Papers” available on the sidebar of this post.)

Dr. Searles book is only one of many in which the use of Coca to treat and cure epileptic seizures is covered, but here is what Dr. Searles had to say:

“Coca Leaf & Acute Disease”

“The relations of Coca to acute disease are extremely important. As a physician, I would not be without it under any consideration. How thoroughly will every physician, understand me when I say that we are not seldom compelled to stand by and witness the death of patients who are really better of the disease which destroys them than perhaps at any previous time during their sickness. We are unable to support them, and they die from exhaustion of the vital forces.”

“But in Coca we have a powerful agent, whose disturbing influence over physiological processes is so little felt that it neither interferes with recovery from disease by natural course, nor with the action of remedies. And its sustaining power is so marvelous, that I prophesy that by its help we shall hereafter be able to cure many cases of disease which were otherwise hopeless.”

“I am informed by my colleague, Dr. John L. Moffat, of Brooklyn, that he has had very encouraging results from the use of Coca in hay fever in four instances. Of course, its action here is antipathic, or rather, it probably acts simply by its sustaining power, and by its antipathic relations to asthma. But even an efficient palliative, which can do no harm, will be welcomed by those who are annually visited by this plague.”

“It has been affirmed by some English authorities that Coca is valueless in epilepsy. For myself I can report that, in one instance of the fully-fledged disease, occurring in a middle-aged lady, but in whom the paroxysms did not recur oftener than once in six months, an apparent cure has been effected by means of Coca alone. She has now passed eighteen months without a seizure. I have also more striking reports from some of the members of this society, who report very marked results in several severe cases which would yield to no other remedy.”

“It is too early yet, however, to claim for Coca really curative powers in this terrible disease, which has so long been an “opprobrium medicorum”. Still, it is highly probable that the forms of it used by the English physicians in their trials were inert. This is rendered more than likely by the fact that one of the most expert chemists of New York City carefully searched both France and England during the summer of 1880 for good Coca, and was unable to obtain a single valuable specimen.”

“In view of the fact that all the drugs now ranked as anti-epileptic by the allopathic school of medicine are so injurious to the general health, and in view of the results attained by myself and my colleagues, imperfect as yet though they are, I earnestly urge the faithful trial of Coca in epilepsy.”

Well, about 140 years have passed and where are those “faithful trials” of Coca Leaf for Epilepsy – and incidentally for Dementia/Alzheimer’s, Congestive Heart Failure, Diabetes, Obesity, and a couple of dozen other killer diseases? Nowhere in sight.

Researchers with Ph.D’s and major institutions behind them are getting tens of millions of dollars to “study” Dementia/Alzheimer’s, but not a peep out of the research establishment about Coca Leaf Tea. And, of course, since I’m not in the club I can’t get a grant, even though I could pretty much prove or disprove the efficacy and safety of Coca leaf for Dementia/Alzheimer’s with a few thousand bucks. But as noted, I don’t have a Ph.D. and I’m not a member of the club, so no institution gives a shit what I say.

Hell, maybe I’ll just do a GoFundMe request for a couple of tickets to Bolivia, grab a hundred kilos or so of fresh Coca Leaf, and come home and start handing out Coca Leaf Tea at a church social or two, and maybe a local nursing home. Think I would get past US Customs/DEA? Might actually be a great idea – let them bust me for trying to bring Coca Leaf in for Dementia/Alzheimer’s patients.

I wonder how many members of the US Congress, who make the laws forbidding Coca Leaf coming into the US, have someone in their family with Dementia/Alzheimer’s. (I’m resisting the obvious snide remark here because while it might be accurate it would also be cruel.)

Anyone have any suggestions?