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Thoughts On Coca, Cannabis, Opium & Tobacco – Gifts Of The Great Spirit

STOPIQOS

IQOS claims an @ 95% reduction in “toxicants” for their new vaporizing device, compared with smoking cigarettes. The claim is that “bake ‘n vape” doesn’t expose users to all the nasty stuff in smoke. I call murderous bullshit.

The “tobacco smoke” all IQOS research compares its vapor to is from rigged industry-supplied “Reference Cigarettes” and by mutual agreement FDA doesn’t look for pesticides in the reference cigarette tobacco contents or smoke or in the vapor from IQOS and Philip Morris, naturally, doesn’t mention them. But unless IQOS tobacco is raised organically, or even responsibly, hazardous pesticides are there, and they are the reason that IQOS vapor is much more toxic than they claim, and would prove to be far more toxic than the smoke from an organic tobacco cigarette if the comparison were to be made, as it soon will be.

What will that do to the IQOS vapor superiority claims, since their device vaporizes what is undoubtedly not organic tobacco? Stay tuned.

Update – Our first pesticide lab test results are in! (12/18) 

So the question that should be before the FDA is – what if it isn’t the Tobacco smoke, or the vapor from Tobacco that’s killing most of the people who die from “smoking-related disease”? What if it isn’t the Tobacco itself at all, but a toxic mix of unregulated pesticides in the smoke or vapor that’s mainly responsible for so much suffering and death? 

OK, if that’s a bit too much, let’s say that it’s mainly the Tobacco, but what if pesticides are adding 100,000 deaths a year to what otherwise would be just 380,000 people instead of the 480,000 souls whose are lives are snuffed out each year purely in pursuit of increased profitability? Would that be worth addressing with some simple regulatory fixes? Are 100,000 lives worth saving? Say that number slowly – one hundred thousand people. Worth the trouble? 

We really do have to ask – what if it’s the insanely toxic pesticide contaminants on the Tobacco that are doing some or maybe even most of the killing, and and what if we could do something about it starting tomorrow, and we don’t?

If you look closely at the data below even if you hate tobacco and all it stands for you may still agree with me that without those contaminants, smoking or vaping Tobacco might still be incredibly unhealthy, and still might be one of the world’s nastiest habits, but it wouldn’t be anywhere nearly as dangerous and lethal without this 100% preventable chemical contamination. Here is new, hard data that demonstrates the widespread presence of extreme pesticide residue contamination in tobacco products.

The IQOS claim of “reduced harm” is deceptive in the extreme because the industry-approved list of nasty stuff, or “toxicants” that are measured when comparing vapor to smoke, deliberately or ‘accidentally’ avoids including an entire set of highly dangerous biologically active components of both tobacco vapor and tobacco smoke – pesticide residues on the tobacco that wind up in the lungs of both smokers and vapers. The pesticides on the tobacco in the IQOS device aren’t going to be degraded or combusted – they are going to be gently vaporized and wafted intact into the lungs, bloodstreams, brains, testicles and ovaries of little IQOS users. That’s more toxicity, not less. A lot more.

Pesticide residues are the Tobacco industry’s dirty secret. Tobacco has been causing widespread sickness and death since the “Green Revolution” gave the industry these xenobiotic chemicals that vastly increase profit by controlling insects, worms, mold and fungus on the tobacco crop without labor costs, and the Tobacco industry has spent literally billions to keep this deadly secret under wraps since 1970 when they pulled the plug on pesticide research worldwide. 

Even before pesticides were developed in the “Green Revolution” that was going to save mankind by feeding everyone, Tobacco was the most heavily contaminated consumable product in the world because growers used every possible “natural” toxin available like heavy metals and plant/mineral poisons used to combat insects, worms, mold and fungus. Smokers have been dying horrible deaths from contaminated Tobacco since right after Europeans “discovered” and expropriated Tobacco and made it the economic foundation of the American colonies. The Tobacco smoked by most of the world for the hundreds of years was so heavily contaminated with poisons like arsenic, mercury and lead compounds used to control bugs and mold that of course it was dangerous to smoke. There was never any such thing as commercial Tobacco without deadly poisons, but those poisons were never included any research into Tobacco-related disease. Of course not – the same doctors who thought that mercury was a dandy medicine thought that tobacco was pure evil, so why would they care about Mercury contamination of Tobacco? The assumption has always been that what was making people sick and killing them was the tobacco, and nothing else mattered. A curious blindness.

So the bottom line is that no European or American physician has ever seen the results of people using uncontaminated Tobacco, but oddly enough none of these doctors have ever questioned whether it was the Tobacco, or its toxic contaminants that was killing people. 

It is an article of faith among American doctors that Tobacco is just plain bad. Full stop. End of story. No need to look further. 

The only possible explanation for the almost universal ignorance of Tobacco contamination and the insane idea that Tobacco itself is so bad that it doesn’t matter what it is contaminated with is that we’re seeing the results of a very expensive, very effective industry disinformation and mind control campaign.

Sure, we can all say with confidence that tobacco smoking causes sickness and death, although nobody really knows exactly how much of that is true and how much is conjecture and bias. But does anyone want to argue over whether maybe 200,000 or more, only 100,000 or so deaths out of the 480,000 “smoking-related” deaths in America could be prevented every year by simple, reasonable pesticide residue standards, enforced with mandatory testing? Do we want to debate how much “smoking-related” sickness and death can be prevented – maybe 200,000 people a year, or “only” 100,000. At what point is it not worth bothering to pass some simple regulations?

That’s why it’s important to call bullshit on IQOS. With IQOS the industry is trying to pivot away from smoking and distract scientists, regulators and the public long enough that they can get some distance from the civil and criminal liability that they would face if legislators and regulators can get their act together in time. 

One HUGE and counter-intuitive problem with the IQOS claim of reduced harm compared with smoking is that “ordinary smokers” actually inhale less pesticide residue than IQOS vapers, who get the whole load of whatever pesticides are contaminating the tobacco they are vaporizing. If anything, smoking is safer than vaping tobacco if you consider take pesticide residue exposure into full account.

Since there is substantial evidence that it may have been more the pesticides than the tobacco itself that has been killing smokers all along, I think it’s reasonable to ask IQOS to include pesticide residue testing in its claims of ‘reduced risk’, don’t you?

This section is dedicated to revealing the deliberate and carefully crafted deceptions that prevent the tobacco industry from being called to full accountability for its cruel exploitation of human weakness.


Part One: Vaporized Pesticides, Public Health & IQOS

Below is a list of the pesticide residues that the tobacco industry’s own science labs know are contaminating the global tobacco supply. At least some of these pesticide residues will be present on every tobacco product. Every tobacco product at the supermarket checkout counter is highly likely to be contaminated with at least some of the pesticides on the following list.

If IQOS is allowed in the US, every cartridge of IQOS tobacco at your supermarket will very likely contaminated unless for some reason Philip Morris International is using organic Tobacco. Could that be what “I Quit Ordinary Smoking” means? No pesticides? Fantastic. No? IQOS isn’t organic Tobacco? Now that would be a real shame because IQOS vapers are going to get the full load of whatever pesticides are there all nicely heated and aerosolized and ready for total absorption.

Which is really criminal, because those pesticides aren’t there because they have to be. They don’t simply ‘occur’ as a natural part of the process. They are only there because manufacturers like PMI decide that there is more profit in having their contract growers use pesticides than in paying them to grow Tobacco organically – which they can do.

The tobacco industry knows how to make profitable organic tobacco products when it wants to and they’ve proved it, so the only reason to use pesticides is because doing so means more profits. It’s simple, and clear. These people aren’t deliberately killing millions of smokers and now vapers. It’s just that they aren’t required to stop using these chemicals, and they have managed to rig the system so their products aren’t subject to normal consumer protection laws, so why not make as much money as possible?

The reason that there’s no public health alarm has to be because the extent of the problem is carefully disguised, right? If it were known, health officials all over the country would be showing up with warrants at the local distributors and minimarts to seize Tobacco products and ship them off to a lab. Right? That would happen, if people knew. Right?

Except – here it is. These pesticide residues are in the world Tobacco supply, which means that at least some of them are highly likely to be in any commercial tobacco product being sold anywhere in the US and worldwide.

This list is current as of 6/18.  (For a post listing pesticides we’re just found (1/19) in five tobacco brands click here)

Table 1.   CPA Guidance Residue Levels

No.

CPA

GRL

(ppm)

Residue definition

Notes

1

2,4,5-T

0.05

2,4,5-T

2

2,4-D

0.2

2,4-D

3

Acephate

0.1

Acephate

4

Acetamiprid

3

Acetamiprid

5

Acibenzolar-S-methyl

5

Acibenzolar-S-methyl

6

Alachlor

0.1

Alachlor

 

7

 

Aldicarb (S)

 

0.5

sum of Aldicarb, Aldicarb sulfoxide and Aldicarb sulfone, expressed as Aldicarb

8

Aldrin + Dieldrin

0.02

Aldrin + Dieldrin

9

Azinphos-ethyl

0.1

Azinphos-ethyl

10

Azinphos-methyl

0.3

Azinphos-methyl

11

Benalaxyl

2

Benalaxyl

12

Benfluralin

0.06

Benfluralin

 

13

 

Benomyl (a)

sum of Benomyl, Carbendazim, and Thiophanate-methyl expressed as Carbendazim

 

see Carbendazim

14

Bifenthrin

3

Bifenthrin

15

Bromophos

0.04

Bromophos

16

Butralin

5

Butralin

17

Camphechlor (S) (Toxaphene)

0.3

Camphechlor (mixture of chlorinated camphenes)

18

Captan

0.7

Captan

19

Carbaryl

0.5

Carbaryl

 

20

 

Carbendazim (a)

 

2

sum of Benomyl, Carbendazim, and Thiophanate-methyl expressed as Carbendazim

 

21

 

Carbofuran (S)

 

0.5

sum of Carbofuran and 3- Hydroxycarbofuran expressed as Carbofuran

22

Chinomethionat

0.1

Chinomethionat

23

Chlorantraniliprole

10

Chlorantraniliprole

24

Chlordane (S)

0.1

sum of cis-Chlordane and trans- Chlordane

25

Chlorfenvinphos (S)

0.04

sum of (E)-Chlorfenvinphos and (Z)-Chlorfenvinphos

No.

CPA

GRL

(ppm)

Residue definition

Notes

26

Chlorothalonil

1

Chlorothalonil

27

Chlorpyrifos

0.5

Chlorpyrifos

28

Chlorpyrifos-methyl

0.2

Chlorpyrifos-methyl

29

Chlorthal-dimethyl

0.5

Chlorthal-dimethyl

30

Clomazone

0.2

Clomazone

31

Cyfluthrin (S)

2

Cyfluthrin (sum of all isomers)

32

Cyhalothrin (S)

0.5

Cyhalothrin (sum of all isomers)

33

Cymoxanil

0.1

Cymoxanil

34

Cypermethrin (S)

1

Cypermethrin (sum of all isomers)

 

35

 

DDT (S)

 

0.2

sum of o,p’- and p,p’-DDT, o,p’-

and p,p’-DDD (TDE), o,p’- and p,p’-DDE expressed as DDT

 

36

 

Deltamethrin (b)

 

1

sum of Deltamethrin and Tralomethrin expressed as Deltamethrin

 

 

37

 

 

Demeton-S-methyl (S)

 

 

0.1

sum of Demeton-S-methyl, Oxydemeton-methyl (Demeton-S- methyl sulfoxide) and Demeton-S- methyl sulfone expressed as Demeton-S-methyl

38

Diazinon

0.1

Diazinon

39

Dicamba

0.2

Dicamba

 

40

 

Dichlorvos (c)

 

0.1

sum of Dichlorvos, Naled and Trichlorfon expressed as Dichlorvos

41

Dicloran

0.1

Dicloran

42

Diflubenzuron

0.1

Diflubenzuron

 

43

 

Dimethoate (d)

 

0.5

sum of Dimethoate and Omethoate expressed as Dimethoate

44

Dimethomorph (S)

2

sum of (E)-Dimethomorph and (Z)-Dimethomorph

 

45

 

Disulfoton (S)

 

0.1

sum of Disulfoton, Disulfoton sulfoxide, and Disulfoton sulfone expressed as Disulfoton

 

 

 

 

 

 

 

 

46

 

 

 

 

 

 

 

 

Dithiocarbamates (as CS2) (e)

 

 

 

 

 

 

 

 

5

 

 

 

 

 

 

 

 

Dithiocarbamates expressed as CS2

In countries where fungal diseases such as blue mould are a persistent problem in the field throughout the growing season, the use of dithio- carbamates (DTC) fungicides may be an essential part of the season-long disease management strategy and in keeping with GAP as a means of ensuring crop quality and economic viability for the producer. Under high disease pressure residues of dithio- carbamates (DTC) fungicides slightly in excess of the specified GRL may be observed.   In countries where there is not a field fungal disease problem the use of fungicides is not necessary, and there should be no residues detected. Consistent with GAP, dithiocarbamates (DTC) fungicides must be used only according to label instructions to combat fungal diseases in the seedbed and in the field.

No.

CPA

GRL

(ppm)

Residue definition

Notes

 

47

 

Endosulfans (S)

 

1

sum of alpha- and beta-isomers and Endosulfan-sulphate expressed as Endosulfan

48

Endrin

0.05

Endrin

49

Ethoprophos

0.1

Ethoprophos

50

Famoxadone

5

Famoxadone

 

51

 

Fenamiphos (S)

 

0.5

sum of Fenamiphos, Fenamiphos sulfoxide and Fenamiphos sulfone expressed as Fenamiphos

52

Fenitrothion

0.1

Fenitrothion

 

53

 

Fenthion (S)

 

0.1

sum of Fenthion, Fenthion sulfoxide and Fenthion sulfone expressed as Fenthion

54

Fenvalerate (S)

1

Fenvalerate (sum of all isomers including Esfenvalerate)

55

Fluazifop-butyl (S)

1

Fluazifop-butyl (sum of all isomers)

56

Flumetralin

5

Flumetralin

57

Fluopyram (g)

5

Fluopyram

58

Folpet

0.2

Folpet

59

HCH (a-, b-, d-)

0.05

HCH (a-, b-, d-)

60

HCH (g-) (Lindane)

0.05

HCH (g-) (Lindane)

 

61

 

Heptachlor (S)

 

0.02

sum of Heptachlor and two Heptachlor epoxides (cis- and trans-) expressed as Heptachlor

62

Hexachlorobenzene

0.02

Hexachlorobenzene

63

Imidacloprid

5

Imidacloprid

64

Indoxacarb (S)

15

Sum of S isomer + R isomer

 

65

 

Iprodione (S)

 

0.5

sum of Iprodione and N-3,5- dichlorophenyl-3-isopropyl-2,4- dioxoimidazolyzin-1-carboxamide expressed as Iprodione

66

Malathion

0.5

Malathion

 

 

 

 

 

67

 

 

 

 

 

Maleic hydrazide

 

 

 

 

 

80

 

 

 

 

Maleic hydrazide (free and bounded form)

In some instances, where GAP is implemented and label recom- mendations with regard to application rates and timing are strictly adhered to, residue levels may exceed the current GRL of 80 ppm as a result of extreme weather conditions and the current technology available for application. However, as with all CPAs, all efforts should be made to strictly follow label application rates, and use should be no more than necessary to achieve the desired effect.

68

Metalaxyl (S)

2

sum of all isomers including Metalaxyl-M / Mefenoxam

69

Methamidophos

1

Methamidophos

70

Methidathion

0.1

Methidathion

 

71

 

Methiocarb (S)

 

0.2

sum of Methiocarb, Methiocarb sulfoxide, and Methiocarb sulfone expressed as Methiocarb

No.

CPA

GRL

(ppm)

Residue definition

Notes

 

72

 

Methomyl (f)

 

1

sum of Methomyl, Methomyl- oxime, and Thiodicarb expressed as Methomyl

73

Methoxychlor

0.05

Methoxychlor

74

Mevinphos (S)

0.04

Mevinphos (sum E and Z isomers)

75

Mirex

0.08

Mirex

76

Monocrotophos

0.3

Monocrotophos

 

77

 

Naled (c)

sum of Dichlorvos, Naled, and Trichlorfon expressed as Dichlorvos

 

see Dichlorvos

78

Nitrofen

0.02

Nitrofen

79

Omethoate (d)

sum of Dimethoate and Omethoate expressed as Dimethoate

see Dimethoate

80

Oxadixyl

0.1

Oxadixyl

81

Oxamyl

0.5

Oxamyl

82

Parathion (-ethyl)

0.06

Parathion

83

Parathion-methyl

0.1

Parathion-methyl

84

Pebulate

0.5

Pebulate

85

Penconazole

1

Penconazole

86

Pendimethalin

5

Pendimethalin

87

Permethrin (S)

0.5

Permethrin (sum of all isomers)

88

Phorate

0.05

Phorate

89

Phosalone

0.1

Phosalone

90

Phosphamidon (S)

0.05

Phosphamidon (sum of E and Z isomers)

91

Phoxim

0.5

Phoxim

92

Piperonyl butoxide

3

Piperonyl butoxide

93

Pirimicarb

0.5

Pirimicarb

94

Pirimiphos-methyl

0.1

Pirimiphos-methyl

95

Profenofos

0.1

Profenofos

96

Propoxur

0.1

Propoxur

97

Pymetrozine

1

Pymetrozine

 

98

 

Pyrethrins (S)

 

0.5

sum of Pyrethrins 1, Pyrethrins 2,

Cinerins 1, Cinerins 2, Jasmolins 1

and Jasmolins 2

99

Tefluthrin

0.1

Tefluthrin

 

100

 

Terbufos (S)

 

0.05

sum of Terbufos, Terbufos sulfoxide and Terbufos sulfone expressed as Terbufos

101

Thiamethoxam

5

Thiamethoxam

 

102

 

Thiodicarb (f)

sum of Methomyl, Methomyl- oxime, and Thiodicarb expressed as Methomyl

 

see Methomyl

103

Thionazin

0.04

Thionazin

 

104

 

Thiophanate-methyl (a)

sum of Benomyl, Carbendazim, and Thiophanate-methyl expressed as Carbendazim

 

see Carbendazim

No.

CPA

GRL

(ppm)

Residue definition

Notes

 

105

 

Tralomethrin (b)

sum of Deltamethrin and Tralomethrin expressed as Deltamethrin

 

see Deltamethrin

 

106

 

Trichlorfon (c)

sum of Dichlorvos, Naled, and Trichlorfon expressed as Dichlorvos

 

see Dichlorvos

107

Trifluralin

0.1

Trifluralin

  • Carbendazim is the degradation product of Benomyl and Thiophanate-methyl. In the case the same sample contains residues of both Carbendazim and/or Benomyl/Thiophanate-methyl, the sum of the residues should not exceed 2

  • Deltamethrin is the degradation product of Tralomethrin. In the case the same sample contains residues of both Deltamethrin and Tralomethrin, the sum of the two residues should not exceed 1

  • Dichlorvos is the degradation product   of  Naled  and     In the case the same sample contains residues of both Dichlorvos and/or Naled/Trichlorfon, the sum of the residues should not exceed 0.1 ppm.

  • Omethoate is the degradation product of Dimethoate. In the case the same sample contains residues of both Dimethoate and Omethoate, the sum of the two residues should not exceed 0.5

  • The Dithiocarbamates Group includes the EBDCs: Mancozeb, Maneb, Metiram, Nabam and Zineb – as well as Amobam, Ferbam, Policarbamate, Propineb, Thiram and

  • Methomyl is the degradation product of Thiodicarb. In the case the same sample contains residues of both Methomyl and Thiodicarb, the sum of the two residues should not exceed 1

  • Fluopyram added to GRL list June 2016